Impaired mitochondrial reserve energy capacity underlies pathology in a number of disease conditions. This impairment can result in vulnerability to tissue damage and organ dysfunction, particularly those with high energy demands, such as the brain, heart, muscles, kidneys and retinas. Wellmond scientists have devised agents to amplify mitochondrial reserve energy capacity, regardless of the specific molecular defects or conditions that impair mitochondrial function. These agents, tailored to specific disease conditions, can be applied to several important categories of Energy
Deficit Disorders: